Basic and clinical aspects of veterinary immunology by Bennie I Osburn; Ronald D Schultz

By Bennie I Osburn; Ronald D Schultz

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Some of these signs include (1) onset of infections in the first 6 weeks of life, suggesting inadequate transfer of maternal Ig; (2) repeated infections that respond poorly to standard therapy; (3) increased susceptibility to low-grade patho­ gens; (4) infection with organisms rarely observed in immunocompetent animals, such as the protozoan Pnenmocystis carinii; (5) systemic ill­ ness following vaccination with live virus vaccines; and (6) marked deficiencies in leukocyte levels, particularly profound neutropenia or lymphopenia of several days duration.

1978). A limitation in this procedure is the time required for sensitization and testing before an answer is obtained. In the case of DNCB, a period of 2 to 3 weeks is usually required. Re- E rosette Bovine Feline Canine Ovine Porcine E rosette SIg EAC rosette Equine — SIg _ + + SIg E rosette - + - - + + + + - ? — EAC rosette - + + + + + + + — + _ + — + B cells Reactive cells T cells EAC rosette SIg E rosette SIg E rosette SIg Marker Species TABLE I I I Guinea pig rbc Bind to monocytes Human, guinea pig rbc Bind to monocytes and neutrophils Sheep rbc Bind to monocytes and neutrophils Sheep rbc Guinea pig (rbc) Comments References Onions (1975) ; Holmberg et al.

Additional studies questioning the role of corticosteroids (Magnuson, 1978) or zinc deficiency (Perryman, unpublished ob­ servations) indicate these factors are not involved in the pathogenesis of CID in horses. , 1979). These studies have met with limited success, largely because of insufficient knowledge of the ontological development of equine fetal immune responses. I M M U N E DEFICIENCIES OF DOMESTIC ANIMALS b. , 1979). Both horses were males, suggesting a sexlinked mode of inheritance. Diagnosis was established on the basis of normal T-lymphocyte function but absence of B lymphocytes.

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